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Bacterial addiction module toxin Doc inhibits translation elongation through its association with the 30S ribosomal subunit
Bacterial toxin–antitoxin (TA) systems (or “addiction modules”) typically facilitate cell survival during intervals of stress by inducing a state of reversible growth arrest. However, upon prolonged stress, TA toxin action leads to cell death. TA systems have also been implicated in several clinical...
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| Main Authors: | , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
National Academy of Sciences
2008
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2311363/ https://ncbi.nlm.nih.gov/pubmed/18398006 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0711949105 |
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