Mutations in XPA that prevent association with ERCC1 are defective in nucleotide excision repair.
The human repair proteins XPA and ERCC1 have been shown to be absolutely required for the incision step of nucleotide excision repair, and recently we identified an interaction between these two proteins both in vivo and in vitro (L. Li, S. J. Elledge, C. A. Peterson, E. S. Bales, and R. J. Legerski...
保存先:
| 出版年: | Mol Cell Biol |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Taylor & Francis
1995
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC230426/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7891694/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.15.4.1993 |
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