Cytoskeletal disrupting agents prevent calmodulin kinase, IQ domain and voltage-dependent facilitation of L-type Ca(2+) channels

A calmodulin (CaM) binding ‘IQ’ domain on the L-type Ca(2+) channel (LTCC) C terminus and calmodulin kinase II (CaMK) both signal increases in LTCC opening probability (P(o)) by shifting LTCCs into a gating mode (mode 2) with long openings through a process called facilitation. However, the mechanis...

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Detalhes bibliográficos
Main Authors: Dzhura, Igor, Wu, Yuejin, Colbran, Roger J, Corbin, Jackie D, Balser, Jeffrey R, Anderson, Mark E
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2002
Assuntos:
Original Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2290681/
https://ncbi.nlm.nih.gov/pubmed/12456820
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2002.021881
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