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Hypoxia potentiates exocytosis and Ca(2+) channels in PC12 cells via increased amyloid β peptide formation and reactive oxygen species generation
Exposure of PC12 cells to chronic hypoxia (CH; 10 % O(2), 24 h) augments catecholamine secretion via formation of a Cd(2+)-resistant Ca(2+) influx pathway, and up-regulates native L-type Ca(2+) channels. These effects are mimicked by exposure of cells to Alzheimer's disease-associated amyloid β...
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| Main Authors: | , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Blackwell Science Inc
2002
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2290370/ https://ncbi.nlm.nih.gov/pubmed/12068059 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2002.017582 |
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