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Exercising skeletal muscle blood flow in humans responds to reduction in arterial oxyhaemoglobin, but not to altered free oxygen
1. We hypothesised that reducing arterial oxyhaemoglobin (O(2)Hb(a)) with carbon monoxide (CO) in both normoxia and hyperoxia, or acute hypoxia would cause similar compensatory increases in human skeletal muscle blood flow and vascular conductance during submaximal exercise, despite vast differences...
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| Hauptverfasser: | , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Blackwell Science Inc
2001
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2278413/ https://ncbi.nlm.nih.gov/pubmed/11208980 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0331l.x |
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