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Fas ligand exerts its pro-inflammatory effects via neutrophil recruitment but not activation
Fas ligand (FasL) expression induces apoptosis of activated T cells and has been suggested as a strategy to inhibit graft rejection. Unfortunately, the use of FasL to confer ‘immune privilege’ in this setting has been hampered by the finding that it may also provoke a destructive granulocytic respon...
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| Autors principals: | , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Blackwell Publishing Inc
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2265864/ https://ncbi.nlm.nih.gov/pubmed/17233740 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1365-2567.2006.02504.x |
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