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Synaptically released glutamate reduces γ-aminobutyric acid (GABA)ergic inhibition in the hippocampus via kainate receptors

Exogenous application of agonists at the kainate subtype of glutamate receptors has been shown to depress evoked monosynaptic inhibition by γ-aminobutyric acid (GABA)ergic interneurons in the hippocampus. This observation has led to the hypothesis that synaptic release of endogenous glutamate might...

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Veröffentlicht in:Proc Natl Acad Sci U S A
Hauptverfasser: Min, Ming-Yuan, Melyan, Zare, Kullmann, Dimitri M.
Format: Artigo
Sprache:Inglês
Veröffentlicht: National Academy of Sciences 1999
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Online-Zugang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC22313/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10449797/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.96.17.9932
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