CB(2)-receptor stimulation attenuates TNF-α-induced human endothelial cell activation, transendothelial migration of monocytes, and monocyte-endothelial adhesion

Targeting cannabinoid-2 (CB(2)) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms by which CB(2) activation exerts its anti-inflammatory effects and the cellular targets are elusive. Here, we investigated the effects of CB...

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Autores principales: Rajesh, Mohanraj, Mukhopadhyay, Partha, Bátkai, Sándor, Haskó, György, Liaudet, Lucas, Huffman, John W., Csiszar, Anna, Ungvari, Zoltan, Mackie, Ken, Chatterjee, Subroto, Pacher, Pál
Formato: Artigo
Lenguaje:Inglês
Publicado: 2007
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2229632/
https://ncbi.nlm.nih.gov/pubmed/17660390
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00688.2007
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