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Endoglin inhibits prostate cancer motility via activation of the ALK2-Smad1 pathway

Endoglin is a transforming growth factor β (TGFβ) superfamily auxiliary receptor. We had previously shown that it suppressed prostate cancer (PCa) cell motility, and that its expression was lost during PCa progression. The mechanism by which endoglin inhibits PCa cell motility is unknown. Here we de...

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Detalles Bibliográficos
Main Authors: Craft, CS, Romero, D, Vary, CPH, Bergan, RC
Formato: Artigo
Idioma:Inglês
Publicado: 2007
Assuntos:
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2199239/
https://ncbi.nlm.nih.gov/pubmed/17496924
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.onc.1210533
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