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Endoglin inhibits prostate cancer motility via activation of the ALK2-Smad1 pathway
Endoglin is a transforming growth factor β (TGFβ) superfamily auxiliary receptor. We had previously shown that it suppressed prostate cancer (PCa) cell motility, and that its expression was lost during PCa progression. The mechanism by which endoglin inhibits PCa cell motility is unknown. Here we de...
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| Main Authors: | , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2007
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2199239/ https://ncbi.nlm.nih.gov/pubmed/17496924 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.onc.1210533 |
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