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Immunoglobulin G–mediated Inflammatory Responses Develop Normally in Complement-deficient Mice

The role of complement in immunoglobulin G–triggered inflammation was studied in mice genetically deficient in complement components C3 and C4. Using the reverse passive Arthus reaction and experimental models of immune hemolytic anemia and immune thrombocytopenia, we show that these mice have types...

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Détails bibliographiques
Auteurs principaux: Sylvestre, Diana, Clynes, Raphael, Ma, Minga, Warren, Henry, Carroll, Michael C., Ravetch, Jeffrey V.
Format: Artigo
Langue:Inglês
Publié: The Rockefeller University Press 1996
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2196366/
https://ncbi.nlm.nih.gov/pubmed/8976192
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