CD36 Mediates the Innate Host Response to β-Amyloid

Accumulation of inflammatory microglia in Alzheimer's senile plaques is a hallmark of the innate response to β-amyloid fibrils and can initiate and propagate neurodegeneration characteristic of Alzheimer's disease (AD). The molecular mechanism whereby fibrillar β-amyloid activates the infl...

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Bibliographic Details
Main Authors: El Khoury, Joseph B., Moore, Kathryn J., Means, Terry K., Leung, Josephine, Terada, Kinya, Toft, Michelle, Freeman, Mason W., Luster, Andrew D.
Format: Artigo
Language:Inglês
Published: The Rockefeller University Press 2003
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Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC2193948/
https://ncbi.nlm.nih.gov/pubmed/12796468
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20021546
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