Tumor necrosis factor/cachectin is an effector of skin and gut lesions of the acute phase of graft-vs.-host disease

Lignende værker

• Cachectin/tumor necrosis factor regulates hepatic acute-phase gene expression.
  af: Perlmutter, D H, et al.
  Udgivet: (1986)
• Bilateral Areolar Lesions in a Patient With Acute Cutaneous Graft-vs-Host Disease
  af: Brunengraber, Lisa N., et al.
  Udgivet: (2011)
• Endogenous tumor necrosis factor (cachectin) is essential to host resistance against Listeria monocytogenes infection.
  af: Nakane, A, et al.
  Udgivet: (1988)
• Granulysin-Bearing Cells in the Skin Lesions of Acute Graft-versus-Host Disease: Possible Mechanisms for Hypohidrosis in Graft-versus-Host Disease
  af: Furudate, Sadanori, et al.
  Udgivet: (2013)
• Metabolic effects of cachectin/tumor necrosis factor are modified by site of production. Cachectin/tumor necrosis factor-secreting tumor in skeletal muscle induces chronic cachexia, while implantation in brain induces predominantly acute anorexia.
  af: Tracey, K J, et al.
  Udgivet: (1990)