Genetic regulation of delayed-type hypersensitivity responses to poly(LTyr,LGlu)-poly(DLAla)--poly(LLys). II. Evidence for a T-T-cell collaboration in delayed-type hypersensitivity responses and for a T- cell defect at the efferent phase in nonresponder H-2k mice

Samankaltaisia teoksia

• Elicitation of delayed-type hypersensitivity responses to poly(L- Tyr,LGlu)-poly(DLAla)--poly(LLys) by anti-idiotypic antibodies
  Julkaistu: (1980)
• Genetic regulation of delayed-type hypersensitivity responses to poly(LTyr,LGu)-poly(DLAla)--poly(LLys). I. Expression of the genetic defect at two phases of the immune process
  Julkaistu: (1980)
• Efficient genetically controlled formation of antibody to a synthetic antigen [poly(LTyr, LGlu)-poly(DLAla)- -poly(LLys)] covalently bound to a synthetic adjuvant (N-acetylmuramyl-L-alanyl-D-isoglutamine).
  Tekijä: Mozes, E, et al.
  Julkaistu: (1980)
• Genetic control of immune response. The dose of antigen given in aqueous solution is critical in determining which mouse strain is high responder to poly(LTyr, LGlu)-poly(LPro)--poly(LLys)
  Julkaistu: (1975)
• Functional helper activity of monoclonal T cell populations: antigen-specific and H-2 restricted cloned T cells provide help for in vitro antibody responses to trinitrophenyl-poly(LTyr,Glu)-poly(DLAla)--poly(LLys).
  Tekijä: Hodes, R J, et al.
  Julkaistu: (1981)