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Proteomics of Transformed Lymphocytes from a Family with Familial Pulmonary Arterial Hypertension

Rationale: Not all family members with BMPR2 mutations develop pulmonary arterial hypertension (PAH), implying that additional modifier genes or proteins are necessary for full expression of the disease. Objectives: To determine whether protein expression is altered in patients with familial PAH (FP...

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Detalhes bibliográficos
Main Authors: Meyrick, Barbara O., Friedman, David B., Billheimer, D. Dean, Cogan, Joy D., Prince, Melissa A., Phillips, John A., Loyd, James E.
Formato: Artigo
Idioma:Inglês
Publicado em: American Thoracic Society 2008
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2176118/
https://ncbi.nlm.nih.gov/pubmed/17932379
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1164/rccm.200703-499OC
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