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Complement C3a Regulates Muc5ac Expression by Airway Clara Cells Independently of Th2 Responses

Rationale: The factors that control the secretion of epithelial mucins are essential to understanding obstructive airway diseases such as asthma. Although the complement anaphylatoxin C3a and its receptor have been shown to promote many features of allergic lung inflammation, the contribution to muc...

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Détails bibliographiques
Auteurs principaux: Dillard, Patricia, Wetsel, Rick A., Drouin, Scott M.
Format: Artigo
Langue:Inglês
Publié: American Thoracic Society 2007
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2176092/
https://ncbi.nlm.nih.gov/pubmed/17400733
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1164/rccm.200701-049OC
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