Hyperammonaemia in V1a vasopressin receptor knockout mice caused by the promoted proteolysis and reduced intrahepatic blood volume

An analysis of arginine-vasopressin (AVP) V1a receptor-deficient (V1aR −/−) mice revealed that glucose homeostasis and lipid metabolism were altered in the mutant mice. Here, we used V1aR −/− mice to investigate whether the deficiency of the V1a receptor, which led to altered insulin sensitivity, af...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Hiroyama, Masami, Aoyagi, Toshinori, Fujiwara, Yoko, Oshikawa, Sayuri, Sanbe, Atsushi, Endo, Fumio, Tanoue, Akito
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Blackwell Publishing Ltd 2007
Aiheet:
Respiratory
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2170841/
https://ncbi.nlm.nih.gov/pubmed/17379633
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.129569
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