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Hyperammonaemia in V1a vasopressin receptor knockout mice caused by the promoted proteolysis and reduced intrahepatic blood volume
An analysis of arginine-vasopressin (AVP) V1a receptor-deficient (V1aR −/−) mice revealed that glucose homeostasis and lipid metabolism were altered in the mutant mice. Here, we used V1aR −/− mice to investigate whether the deficiency of the V1a receptor, which led to altered insulin sensitivity, af...
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Main Authors: | , , , , , , |
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Formato: | Artigo |
Idioma: | Inglês |
Publicado em: |
Blackwell Publishing Ltd
2007
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Assuntos: | |
Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2170841/ https://ncbi.nlm.nih.gov/pubmed/17379633 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.129569 |
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