Isolation of full-length ATM cDNA and correction of the ataxia-telangiectasia cellular phenotype

A gene mutated in the human genetic disorder ataxia-telangiectasia (A-T), ATM, was recently identified by positional cloning. ATM is a member of the phosphatidylinositol-3-kinase superfamily, some of which are protein kinases and appear to have important roles in cell cycle control and radiation sig...

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Veröffentlicht in:	Proc Natl Acad Sci U S A
Hauptverfasser:	Zhang, Ning, Chen, Philip, Khanna, Kum Kum, Scott, Shaun, Gatei, Magtouf, Kozlov, Sergei, Watters, Dianne, Spring, Kevin, Yen, Tim, Lavin, Martin F.
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	National Academy of Sciences 1997
Schlagworte:	Biological Sciences
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC21549/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9223307/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.94.15.8021
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Isolation of full-length ATM cDNA and correction of the ataxia-telangiectasia cellular phenotype

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