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Altered trophoblast proliferation is insufficient to account for placental dysfunction in Egfr null embryos
Homozygosity for the Egfr(tm1Mag) null allele in mice leads to genetic background dependent placental abnormalities and embryonic lethality. Molecular mechanisms or genetic modifiers that differentiate strains with surviving versus non-surviving Egfr nullizygous embryos have yet to be identified. Eg...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2121666/ https://ncbi.nlm.nih.gov/pubmed/17822758 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.placenta.2007.07.005 |
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