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Mutation in pre-mRNA adenosine deaminase markedly attenuates neuronal tolerance to O(2) deprivation in Drosophila melanogaster

O(2) deprivation can produce many devastating clinical conditions such as myocardial infarct and stroke. The molecular mechanisms underlying the inherent tissue susceptibility or tolerance to O(2) lack are, however, not well defined. Since the fruit fly, Drosophila melanogaster, is extraordinarily t...

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Hlavní autoři: Ma, Enbo, Gu, Xiang-Qun, Wu, Xiaohui, Xu, Tian, Haddad, Gabriel G.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Clinical Investigation 2001
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC208948/
https://ncbi.nlm.nih.gov/pubmed/11254668
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