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Multiple cardiovascular defects caused by the absence of alternatively spliced segments of fibronectin

Alternatively spliced variants of fibronectin (FN) containing exons EIIIA and EIIIB are expressed around newly forming vessels in development and disease but are downregulated in mature vasculature. The sequences and patterns of expression of these splice variants are highly conserved among vertebra...

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Bibliografske podrobnosti
Main Authors: Astrof, Sophie, Crowley, Denise, Hynes, Richard O.
Format: Artigo
Jezik:Inglês
Izdano: 2007
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC2080666/
https://ncbi.nlm.nih.gov/pubmed/17706958
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ydbio.2007.07.005
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