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Multiple cardiovascular defects caused by the absence of alternatively spliced segments of fibronectin
Alternatively spliced variants of fibronectin (FN) containing exons EIIIA and EIIIB are expressed around newly forming vessels in development and disease but are downregulated in mature vasculature. The sequences and patterns of expression of these splice variants are highly conserved among vertebra...
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| Main Authors: | , , |
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| Format: | Artigo |
| Language: | Inglês |
| Published: |
2007
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2080666/ https://ncbi.nlm.nih.gov/pubmed/17706958 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ydbio.2007.07.005 |
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