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Attenuation of store-operated Ca(2+) current impairs salivary gland fluid secretion in TRPC1(−/−) mice
Agonist-induced Ca(2+) entry via store-operated Ca(2+) (SOC) channels is suggested to regulate a wide variety of cellular functions, including salivary gland fluid secretion. However, the molecular components of these channels and their physiological function(s) are largely unknown. Here we report t...
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| Asıl Yazarlar: | , , , , , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
National Academy of Sciences
2007
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2077292/ https://ncbi.nlm.nih.gov/pubmed/17956991 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0701254104 |
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