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Pulmonary inflammation induced by repeated inhalations of beta(1,3)-D-glucan and endotoxin.

In an animal model of hypersensitivity pneumonitis (HP) guinea-pigs were exposed for 5 weeks to an aerosol of bacterial endotoxin, beta(1,3)-D-glucan (curdlan) or a combination. Exposure to endotoxin or curdlan showed only small changes in inflammatory cells in airways or the lung wall, histological...

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Bibliographische Detailangaben
Hauptverfasser: Fogelmark, B., Sjöstrand, M., Rylander, R.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 1994
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2002110/
https://ncbi.nlm.nih.gov/pubmed/8199009
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