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UNC-1 regulates gap junctions important to locomotion in C. elegans
In C. elegans, loss-of-function (lf) mutations of the stomatin-like protein (SLP) UNC-1 and the innexin UNC-9 inhibit locomotion [1, 2] and modulate sensitivity to volatile anesthetics [3, 4]. It was unknown why unc-1(lf) and unc-9(lf) mutants have similar phenotypes. We tested the hypothesis that U...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1976340/ https://ncbi.nlm.nih.gov/pubmed/17658257 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cub.2007.06.060 |
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