Predominant activation of MAP kinases and pro‐destructive/pro‐inflammatory features by TNF α in early‐passage synovial fibroblasts via TNF receptor‐1: failure of p38 inhibition to suppress matrix metalloproteinase‐1 in rheumatoid arthritis

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• Prostaglandin E(2) Differentially Modulates Proinflammatory/Prodestructive Effects of TNF-α on Synovial Fibroblasts via Specific E Prostanoid Receptors/cAMP()
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