Helicobacter pylori Chemotaxis Modulates Inflammation and Bacterium-Gastric Epithelium Interactions in Infected Mice

The ulcer-causing pathogen Helicobacter pylori uses directed motility, or chemotaxis, to both colonize the stomach and promote disease development. Previous work showed that mutants lacking the TlpB chemoreceptor, one of the receptors predicted to drive chemotaxis, led to less inflammation in the ge...

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Tallennettuna:
Bibliografiset tiedot
Päätekijät: Williams, Susan M., Chen, Yu-Ting, Andermann, Tessa M., Carter, J. Elliot, McGee, David J., Ottemann, Karen M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Microbiology 2007
Aiheet:
Host Response and Inflammation
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC1952010/
https://ncbi.nlm.nih.gov/pubmed/17517875
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.00082-07
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