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Toll-like receptor—mediated cytokine production is differentially regulated by glycogen synthase kinase 3

The cellular mechanisms that directly regulate the inflammatory response after Toll-like receptor (TLR) stimulation are unresolved at present. Here we report that glycogen synthase kinase 3 (GSK3) differentially regulates TLR-mediated production of pro- and anti-inflammatory cytokines. Stimulation o...

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Bibliografiset tiedot
Päätekijät: Martin, Michael, Rehani, Kunal, Jope, Richard S, Michalek, Suzanne M
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2005
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC1933525/
https://ncbi.nlm.nih.gov/pubmed/16007092
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ni1221
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