Human cytomegalovirus IE2 86-kilodalton protein binds p53 but does not abrogate G1 checkpoint function.
Physical interactions between human cytomegalovirus (HCMV) immediate-early (IE) proteins and key cell cycle regulatory proteins have been suggested as a mechanism whereby this herpesvirus modifies cellular control of proliferation. Observed similarities to interactions of other DNA virus proteins (h...
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| Опубліковано в:: | J Virol |
|---|---|
| Автори: | , |
| Формат: | Artigo |
| Мова: | Inglês |
| Опубліковано: |
American Society for Microbiology (ASM)
1997
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| Предмети: | |
| Онлайн доступ: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191841/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9223475/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.8.5861-5870.1997 |
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