Modification of the Sendai virus-specific antibody and CD8+ T-cell responses in mice homozygous for disruption of the interleukin-4 gene.

Homozygous disruption (-/-) of the interleukin-4 (IL-4) gene did not obviously modify the severity of Sendai virus infection in the highly susceptible 129/J mouse strain. The virus was cleared from the respiratory tract, and potent cytotoxic T lymphocyte (CTL) effectors were present in the cell popu...

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Bibliografiska uppgifter

I publikationen:	J Virol
Huvudupphov:	Mo, X Y, Sangster, M Y, Tripp, R A, Doherty, P C
Materialtyp:	Artigo
Språk:	Inglês
Utgiven:	American Society for Microbiology (ASM) 1997
Ämnen:	Research Article
Länkar:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191366/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9032393/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.3.2518-2521.1997
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