Attenuation of endotoxin-induced multiple organ dysfunction by 1-amino-2-hydroxy-guanidine, a potent inhibitor of inducible nitric oxide synthase.

Ähnliche Einträge

• The multiple organ dysfunction syndrome caused by endotoxin in the rat: attenuation of liver dysfunction by inhibitors of nitric oxide synthase.
  von: Thiemermann, C., et al.
  Veröffentlicht: (1995)
• Isothioureas: potent inhibitors of nitric oxide synthases with variable isoform selectivity.
  von: Southan, G J, et al.
  Veröffentlicht: (1995)
• Attenuation of the induction of nitric oxide synthase by endogenous glucocorticoids accounts for endotoxin tolerance in vivo.
  von: Szabó, C, et al.
  Veröffentlicht: (1994)
• Beneficial effects and improved survival in rodent models of septic shock with S-methylisothiourea sulfate, a potent and selective inhibitor of inducible nitric oxide synthase.
  von: Szabó, C, et al.
  Veröffentlicht: (1994)
• Nitric oxide-mediated hyporeactivity to noradrenaline precedes the induction of nitric oxide synthase in endotoxin shock.
  von: Szabó, C., et al.
  Veröffentlicht: (1993)