Abrogation of growth arrest signals by human papillomavirus type 16 E7 is mediated by sequences required for transformation.

Ítems similars

• The ability of human papillomavirus E6 proteins to target p53 for degradation in vivo correlates with their ability to abrogate actinomycin D-induced growth arrest.
  per: Foster, S A, et al.
  Publicat: (1994)
• Growth arrest by induction of p53 in DNA damaged keratinocytes is bypassed by human papillomavirus 16 E7.
  per: Demers, G W, et al.
  Publicat: (1994)
• The E7 gene of human papillomavirus type 16 is sufficient for immortalization of human epithelial cells.
  per: Halbert, C L, et al.
  Publicat: (1991)
• Disruption of the G(1)/S Transition in Human Papillomavirus Type 16 E7-Expressing Human Cells Is Associated with Altered Regulation of Cyclin E
  per: Martin, Larry G., et al.
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• Destabilization of the Retinoblastoma Tumor Suppressor by Human Papillomavirus Type 16 E7 Is Not Sufficient To Overcome Cell Cycle Arrest in Human Keratinocytes
  per: Helt, Anna-Marija, et al.
  Publicat: (2001)