Trypsin activation pathway of rotavirus infectivity.
The infectivity of rotaviruses is increased by and most probably is dependent on trypsin treatment of the virus. This proteolytic treatment specifically cleaves VP4, the protein that forms the spikes on the surface of the virions, to polypeptides VP5 and VP8. This cleavage has been reported to occur...
Uloženo v:
| Vydáno v: | J Virol |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Microbiology (ASM)
1996
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC190599/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8709201/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.70.9.5832-5839.1996 |
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