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Two herpes simplex virus type 1 latency-active promoters differ in their contributions to latency-associated transcript expression during lytic and latent infections.

Herpes simplex virus type 1 (HSV-1) establishes latency in human sensory ganglia, during which time the viral genome is transcriptionally silent with the exception of the latency-associated transcripts (LATs). The most abundant LAT is a 2-kb RNA whose biosynthesis is poorly characterized. The 2-kb L...

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Detalhes bibliográficos
Publicado no:J Virol
Principais autores: Chen, X, Schmidt, M C, Goins, W F, Glorioso, J C
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology (ASM) 1995
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189734/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7494302/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.69.12.7899-7908.1995
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