Stimulation of the cAMP pathway protects cultured cerebellar granule neurons against alcohol-induced cell death by activating the neuronal nitric oxide synthase (nNOS) gene.

Neuronal loss is a key component of fetal alcohol syndrome pathophysiology. Therefore, identification of molecules and signaling pathways that ameliorate alcohol-induced neuronal death is important. We have previously reported that neuronal nitric oxide synthase (nNOS) can protect developing cerebel...

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Hlavní autoři: Karaçay, Bahri, Li, Guiying, Pantazis, Nicholas J., Bonthius, Daniel J.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2007
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC1894830/
https://ncbi.nlm.nih.gov/pubmed/17306238
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2007.01.059
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