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Human immunodeficiency virus type 1 pol gene mutations which cause decreased susceptibility to 2',3'-dideoxycytidine.

To investigate whether human immunodeficiency virus type 1 pol gene mutations are selected during prolonged 2',3'-dideoxycytidine (ddC) therapy, we used the polymerase chain reaction to amplify a portion of the reverse transcriptase segment of the pol gene from the peripheral blood mononuc...

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Detalhes bibliográficos
Main Authors: Fitzgibbon, J E, Howell, R M, Haberzettl, C A, Sperber, S J, Gocke, D J, Dubin, D T
Formato: Artigo
Idioma:Inglês
Publicado em: 1992
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC189244/
https://ncbi.nlm.nih.gov/pubmed/1317143
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