Host response to Sendai virus in mice lacking class II major histocompatibility complex glycoproteins.
The development of Sendai virus-specific cytotoxic T-lymphocyte (CTL) effectors and precursors (CTLp) has been compared for mice that are homozygous (-/-) for a disruption of the H-2I-Ab class II major histocompatibility complex glycoprotein and for normal (+/+) controls. The generation of CD8+ CTLp...
Tallennettuna:
| Julkaisussa: | J Virol |
|---|---|
| Päätekijät: | , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Society for Microbiology (ASM)
1995
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC188729/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7853474/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.69.3.1429-1434.1995 |
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