Induction of different morphologic features of malignant melanoma and pigmented lesions after transformation of murine melanocytes with bFGF-cDNA and H-ras, myc, neu, and E1a oncogenes.

Eitemau Tebyg

• Direct reprogramming by oncogenic Ras and Myc
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  Cyhoeddwyd: (2013)
• Heparin-treated, v-myc-transformed chicken heart mesenchymal cells assume a normal morphology but are hypersensitive to epidermal growth factor (EGF) and brain fibroblast growth factor (bFGF); cells transformed by the v-Ha-ras oncogene are refractory to EGF and bFGF but are hypersensitive to insulin-like growth factors.
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• Behavior of myc and ras oncogenes in transformation of rat embryo fibroblasts.
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• Molecular Analysis of Oncogenes, ras Family Genes (N‐ras, K‐ras, H‐ras), myc Family Genes (c‐myc, N‐myc) and mdm2 in Natural Killer Cell Neoplasms
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  Cyhoeddwyd: (2002)
• Regulation of bFGF gene expression and subcellular distribution of bFGF protein in adrenal medullary cells
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