Murid Herpesvirus 4 Strain 68 M2 Protein Is a B-Cell-Associated Antigen Important for Latency but Not Lymphocytosis

This work describes analyses of the function of the murid herpesvirus 4 strain 68 (MHV-68) M2 gene. A frameshift mutation was made in the M2 open reading frame that caused premature termination of translation of M2 after amino acid residue 90. The M2 mutant showed no defect in productive replication...

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Detalles Bibliográficos

Publicado en:	J Virol
Autores principales:	Macrae, Alastair I., Usherwood, Edward J., Husain, S. Mazher, Flaño, Emilio, Kim, In-Jeong, Woodland, David L., Nash, Anthony A., Blackman, Marcia A., Sample, Jeffery T., Stewart, James P.
Formato:	Artigo
Lenguaje:	Inglês
Publicado:	American Society for Microbiology (ASM) 2003
Materias:	Pathogenesis and Immunity
Acceso en línea:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC187398/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12915582/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.77.17.9700-9709.2003
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