Fibroblasts Isolated from Normal Lungs and Those with Idiopathic Pulmonary Fibrosis Differ in Interleukin-6/gp130-Mediated Cell Signaling and Proliferation

Interleukin (IL)-6 and IL-11 are elevated in a variety of lung conditions and may impact on repair mechanisms in chronic inflammatory disorders. However, the mechanisms by which these cytokines influence fibroblast proliferation in normal and disease states have not been previously addressed. We exa...

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Main Authors: Moodley, Yuben P., Scaffidi, Amelia K., Misso, Neil L., Keerthisingam, Carmel, McAnulty, Robin J., Laurent, Geoff J., Mutsaers, Steven E., Thompson, Philip J., Knight, Darryl A.
格式: Artigo
語言:Inglês
出版: American Society for Investigative Pathology 2003
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC1868172/
https://ncbi.nlm.nih.gov/pubmed/12819039
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