GILZ mediates the antiproliferative activity of glucocorticoids by negative regulation of Ras signaling

Tsc22d3 coding for glucocorticoid-induced leucine zipper (GILZ) was initially identified as a dexamethasone-responsive gene involved in the control of T lymphocyte activation and apoptosis. However, the physiological role of this molecule and its function in the biological activity of glucocorticoid...

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Autores principales: Ayroldi, Emira, Zollo, Ornella, Bastianelli, Alessandra, Marchetti, Cristina, Agostini, Massimiliano, Di Virgilio, Rosa, Riccardi, Carlo
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Clinical Investigation 2007
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Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC1865030/
https://ncbi.nlm.nih.gov/pubmed/17492054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI30724
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