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Pentoxifylline and CD14 antibody additively inhibit priming of polymorphonuclear leukocytes for enhanced release of superoxide by lipopolysaccharide: possible mechanism of these actions.

Lipopolysaccharide (LPS) primes human polymorphonuclear leukocytes (PMN) for enhanced O2- production in response to stimulation by N-formyl-methionyl-leucyl-phenylalanine (fMet-Leu-Phe). Serum factor is essential for priming at lower concentrations of LPS. Complexes of LPS and LPS-binding protein ar...

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Библиографические подробности
Главные авторы: Yasui, K, Komiyama, A, Molski, T F, Sha'afi, R I
Формат: Artigo
Язык:Inglês
Опубликовано: 1994
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC186204/
https://ncbi.nlm.nih.gov/pubmed/7509321
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