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Absence of exogenous interleukin-4-induced apoptosis of gingival macrophages may contribute to chronic inflammation in periodontal diseases.

Inflamed gingival tissues are enriched in macrophages (MOs) and CD4-positive T cells; however, T helper-type cytokines such as interleukin (IL)-2 and IL-4 are absent. Therefore, we investigated whether a relationship exists between IL-4 receptor (IL-4R) expression and MO persistence in the absence o...

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Bibliografiska uppgifter
Huvudupphovsmän: Yamamoto, M., Kawabata, K., Fujihashi, K., McGhee, J. R., Van Dyke, T. E., Bamberg, T. V., Hiroi, T., Kiyono, H.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 1996
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Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC1861600/
https://ncbi.nlm.nih.gov/pubmed/8546223
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