Increased GABAB receptor function in mouse frontal cortex after repeated administration of antidepressant drugs or electroconvulsive shocks.

Lignende værker

• GABAB-receptor mediated inhibition of potassium-evoked release of endogenous 5-hydroxytryptamine from mouse frontal cortex.
  af: Gray, J. A., et al.
  Udgivet: (1987)
• Repeated administration of desipramine and a GABAB receptor antagonist, CGP 36742, discretely up-regulates GABAB receptor binding sites in rat frontal cortex.
  af: Pratt, G. D., et al.
  Udgivet: (1993)
• The effects of single and repeated electroconvulsive shock administration on the release of 5-hydroxytryptamine and noradrenaline from cortical slices of rat brain.
  af: Green, A. R., et al.
  Udgivet: (1987)
• The effect of repeated electroconvulsive shock on GABA synthesis and release in regions of rat brain.
  af: Green, A. R., et al.
  Udgivet: (1987)
• EFFECTS OF CHRONIC ANTIDEPRESSANT DRUG ADMINISTRATION AND ELECTROCONVULSIVE SHOCK ON ACTIVITY OF DOPAMINERGIC NEURONS IN THE VENTRAL TEGMENTUM
  af: West, Charles Hutchison Keesor, et al.
  Udgivet: (2010)