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Ca(V)3.2 is the major molecular substrate for redox regulation of T-type Ca(2+) channels in the rat and mouse thalamus
Although T-type Ca(2+) channels in the thalamus play a crucial role in determining neuronal excitability and are involved in sensory processing and pathophysiology of epilepsy, little is known about the molecular mechanisms involved in their regulation. Here, we report that reducing agents, includin...
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主要な著者: | , , , , , , , |
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フォーマット: | Artigo |
言語: | Inglês |
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Blackwell Science Inc
2006
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主題: | |
オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1817755/ https://ncbi.nlm.nih.gov/pubmed/16644797 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.110395 |
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