Dysfunction of GABA(A) receptor glycolysis-dependent modulation in human partial epilepsy

A reduction in GABAergic neurotransmission has been put forward as a pathophysiological mechanism for human epilepsy. However, in slices of human epileptogenic neocortex, GABAergic inhibition can be clearly demonstrated. In this article we present data showing an increase in the functional lability...

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Bibliografski detalji
Glavni autori: Laschet, Jacques J., Kurcewicz, Irène, Minier, Frédéric, Trottier, Suzanne, Khallou-Laschet, Jamila, Louvel, Jacques, Gigout, Sylvain, Turak, Baris, Biraben, Arnaud, Scarabin, Jean-Marie, Devaux, Bertrand, Chauvel, Patrick, Pumain, René
Format: Artigo
Jezik:Inglês
Izdano: National Academy of Sciences 2007
Teme:
Biological Sciences
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC1805601/
https://ncbi.nlm.nih.gov/pubmed/17360668
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0606451104
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