Dysfunction of GABA(A) receptor glycolysis-dependent modulation in human partial epilepsy

A reduction in GABAergic neurotransmission has been put forward as a pathophysiological mechanism for human epilepsy. However, in slices of human epileptogenic neocortex, GABAergic inhibition can be clearly demonstrated. In this article we present data showing an increase in the functional lability...

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Detalhes bibliográficos
Main Authors: Laschet, Jacques J., Kurcewicz, Irène, Minier, Frédéric, Trottier, Suzanne, Khallou-Laschet, Jamila, Louvel, Jacques, Gigout, Sylvain, Turak, Baris, Biraben, Arnaud, Scarabin, Jean-Marie, Devaux, Bertrand, Chauvel, Patrick, Pumain, René
Formato: Artigo
Idioma:Inglês
Publicado em: National Academy of Sciences 2007
Assuntos:
Biological Sciences
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1805601/
https://ncbi.nlm.nih.gov/pubmed/17360668
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0606451104
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