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Reactive oxygen species and p38 phosphorylation regulate the protective effect of Δ(9) –tetrahydrocannabinol in the apoptotic response to NMDA
NMDA causes oxidative stress in neurons, and produces cell death involving elements of both necrosis and apoptosis. To examine the neuroprotective mechanism of Δ(9)-Tetrahydrocannabinol (THC) in NMDA-induced death of AF5 cells, we measured reactive oxygen species (ROS) formation after exposure to NM...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2005
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1790967/ https://ncbi.nlm.nih.gov/pubmed/16098661 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neulet.2005.07.028 |
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