Tet(M)-promoted release of tetracycline from ribosomes is GTP dependent.
Tet(M) protein, which displays homology to elongation factor G (EF-G), interacts with the protein biosynthetic machinery to render this process resistant to tetracycline in vivo and in vitro. To clarify the basis of the resistance mechanism, the effects of Tet(M) on several reactions which occur dur...
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| Publicado no: | J Bacteriol |
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| Autor principal: | |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Microbiology (ASM)
1996
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC178077/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8655505/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.178.11.3246-3251.1996 |
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