Lipopolysaccharide modulation of normal enterocyte turnover by toll-like receptors is mediated by endogenously produced tumour necrosis factor α

Background: Circulating levels of endotoxin (or lipopolysaccharide (LPS)) and anti-endotoxin antibodies are increased in patients with inflammatory bowel disease, supporting the hypothesis of a role for endogenous bacterial products in the pathogenesis of these disorders. Aim: The aim of this study...

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Autori principali: Ruemmele, F M, Beaulieu, J F, Dionne, S, Levy, E, Seidman, E G, Cerf-Bensussan, N, Lentze, M J
Natura: Artigo
Lingua:Inglês
Pubblicazione: Copyright 2002 by Gut 2002
Soggetti:
Intestinal Infection
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1773452/
https://ncbi.nlm.nih.gov/pubmed/12427787
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