Lack of the extracellular 19-kilodalton fibrinogen-binding protein from Staphylococcus aureus decreases virulence in experimental wound infection.

A mutant deficient for the 19-kDa extracellular fibrinogen-binding protein (Fib) from Staphylococcus aureus has been constructed. The gene was inactivated by allele replacement. A 2.0-kb fragment from transposon Tn4001 carrying the gene for gentamicin resistance was inserted into the gene encoding F...

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Pubblicato in:	Infect Immun
Autori principali:	Palma, M, Nozohoor, S, Schennings, T, Heimdahl, A, Flock, J I
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	American Society for Microbiology (ASM) 1996
Soggetti:	Research Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC174520/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8945578/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.64.12.5284-5289.1996
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