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The histamine H(3) receptor agonist N -methylhistamine produced by Helicobacter pylori does not alter somatostatin release from cultured rabbit fundic D-cells

Background—The mechanisms underlying the suppression of somatostatin dependent reflexes in Helicobacter pylori infection are not fully determined. The H pylori product N -methylhistamine and inflammatory mediators such as tumour necrosis factor-α (TNF-α) may be responsible for the alterations in som...

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Detalles Bibliográficos
Autores principales: Beales, I, Calam, J
Formato: Artigo
Lenguaje:Inglês
Publicado: 1998
Materias:
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC1727208/
https://ncbi.nlm.nih.gov/pubmed/10189840
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